References
- Barbers R. C., Evans M. J., Gong H., Jr., Tashkin D. P. Enhanced alveolar monocyte (macrophage) proliferation in tobacco and marijuana smokers. Am. Rev. Respir. Dis. 1991; 143: 1092–1095
- Bitterman P. B., Saltzmam L. E., Adelberg S., Ferrans V. J., Crystal R. G. Alveolar mononuclear phagocyte population in the chronically inflamed lung. J. Clin. Invest. 1984; 74: 460–469
- Craighead J. E., Mossman B. T. The pathogenesis of asbestos-associated diseases. N. Engl. J. Med. 1982; 306: 1446–1455
- Crystal R. G., Gadek J. E., Ferrans V. J., Fulmer J. D., Line B. R., Hunninghake G. W. Interstitial lung disease: current concept of pathogenesis, staging and therapy. Am. J. Med. 1981; 70: 542–568
- Donaldson K., Brown G. M., Brown D. M., Robertson M. D., Slight J., Cowie H., Jones A. D., Bolton R. E., Davis J. M. G. Contrasting bronchoalveolar leukocyte responses in rats inhaling coal mine dust, quartz, or titanium dioxide: effect of coal rank, airborne mass concentration, and cessation of exposure. Environ. Res. 1990; 52: 62–76
- Evans M. J., Cabral L. T., Stephens R. J., Freeman G. Cell division of alveolar macrophages in rat lung following exposure to NO2. Am. J. Pathol. 1973; 70: 199–208
- Evans M. J., Shami S. G., Martinez L. A. Enhanced proliferation of pulmonary alveolar macrophages after carbon instillation in mice depleted of blood monocytes by strontium-89. Lab. Invest. 1986; 54: 154–159
- Golde D. W., Byers L. A., Finley T. N. Proliferative capacity of human alveolar macrophages. Nature 1974; 247: 373–375
- Hocking W. G., Golde D. W. The pulmonary alveolar macrophage. N. Engl. J. Med. 1979; 301: 639–645
- Kagan E., Oghiso Y., Hartmann D. P. The effects of chrysotile and crocidolite asbestos on the lower respiratory tract: analysis of bronchoalveolar lavage constituents. Environ. Res. 1983a; 32: 382–397
- Kagan E., Oghiso Y., Hartmann D. P. Enhanced release of a chemoattractant for alveolar macrophages after asbestos inhalation. Am. Rev. Respir. Dis. 1983b; 128: 680–687
- Lin H. S., Lokeshwar B. L., Hsu S. Both granulocyte-macrophage CSF and macrophage CSF control the proliferation and survival of the same subset of alveolar macrophages. J. Immunol. 1989; 142: 515–519
- Oghiso Y., Yamada Y., Shibata Y., Yoshida T. Maintenance of radioresistant alveolar colony-forming cells in mice bearing pulmonary foreign-body inflammation after 89Sr-injec-tion. Reg. Immunol. 1991; 3: 318–322
- Sawyer R. T. The significance of local resident pulmonary alveolar macrophage proliferation to population renewal. J. Leukocyte Biol. 1986; 39: 77–87
- Shellito J., Esparza C., Armstrong C. Maintenance of the normal rat alveolar macrophage cell population. The roles of monocyte influx and alveolar macrophage proliferation in situ. Am. Rev. Respir. Dis. 1987; 135: 78–82
- Spurzem J. R., Saltini C., Rom W., Winchester R. J., Crystal R. G. Mechanisms of macrophage accumulation in the lung of asbestos-exposed subjects. Am. Rev. Respir. Dis. 1987; 136: 276–280
- Talbot R. J., Nicholas L., Morgan A., Moores S. R. Effect of inhaled a-emitting nuclides on mouse alveolar macrophages. Radiat. Res. 1989; 119: 271–285
- Tarling J. D., Lin H. S., Hsu S. Self-renewal of pulmonary alveolar macrophages: evidence from radiation chimera studies. J. Leukocyte Biol. 1987; 42: 443–446
- van oud Albas A. B., van Furth R. Origin, kinetics and characteristics of pulmonary macrophages in the normal steady state. J. Exp. Med. 1972; 149: 1504–1518
- van oud Albas A. B., van der Linden-Schrever B., van Furth R. Origin and kinetics of pulmonary macrophages during an inflammatory reaction induced by an intraalveolar administration of aerosolized heat-killed BCG. Am. Rev. Respir. Dis. 1983; 128: 276–281
- Warheit D. B., George G., Hill L. H., Snyderman R., Brody R. Inhaled asbestos activates a complement dependent chemoattractant for macrophages. Lab. Invest. 1985; 52: 505–514