The disulphide cleavage derivative (C42-4) of 11′-deoxyverticillin A (C42) fails to induce apoptosis and genomic instability in HeLa cells

Bolin Houa State Key Laboratory of Mycology, Institute of Microbiology, Chinese Academy of Sciences, Beijing, China;b CAS Key Laboratory of Microbial Physiological and Metabolic Engineering, Institute of Microbiology, Chinese Academy of Sciences, Beijing, ChinaView further author information

Huaiyi Yangb CAS Key Laboratory of Microbial Physiological and Metabolic Engineering, Institute of Microbiology, Chinese Academy of Sciences, Beijing, ChinaView further author information

Erwei Lia State Key Laboratory of Mycology, Institute of Microbiology, Chinese Academy of Sciences, Beijing, China;c Institutional Center for Shared Technologies and Facilities, Institute of Microbiology, Chinese Academy of Sciences, Beijing, ChinaCorrespondence[email protected]
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Xuejun Jianga State Key Laboratory of Mycology, Institute of Microbiology, Chinese Academy of Sciences, Beijing, ChinaCorrespondence[email protected]
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The disulphide cleavage derivative (C42-4) of 11′-deoxyverticillin A (C42) fails to induce apoptosis and genomic instability in HeLa cells

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The disulphide cleavage derivative (C42-4) of 11′-deoxyverticillin A (C42) fails to induce apoptosis and genomic instability in HeLa cells

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